“I’m Getting worse/Doctor retires/no doctors/scared & so confused/no where to go…Feb7/22 MRI before one I ported earlier today😭
Multiple scattered small foci of FLAIR and T2 signal hyperintensity in the bilateral subcortical and deep supratentorial white matter. Small periventricular lesion adjacent to the frontal horn of the left lateral ventricle, axial image 68. No definite juxtacortical lesions. Small lesions in the pons. No lesions in the visualized spinal cord. When compared to the previous, the lesions have increased in number. There is no associated restricted diffusion.
Small linear lesions in the cortex of the left cerebral hemisphere compatible with chronic lacunar infarctions, best appreciated on the T2weighted images, not seen on the previous.
Preserved normal caliber and configuration of ventricles and extraaxial CSF spaces. No intracranial spaceoccupying abnormality. Preserved major intracranial flow voids.
CSF extension to the sella. Stable small pineal gland cyst. Unremarkable orbital structures.
Clear paranasal sinuses and mastoid air cells.
No concerning bone marrow signal abnormality. Page: 1 of 2 IMPRESSION:
Redemonstration of scattered foci of FLAIR and T2 signal
Unremarkable orbital structures.
Clear paranasal sinuses and mastoid air cells.
No concerning bone marrow signal abnormality. IMPRESSION:
Redemonstration of scattered foci of FLAIR and T2 signal hyperintensity within the bilateral supratentorial white matter, increased in number since the comparison MR. Primary differential considerations include early onset of microangiopathic disease and demyelination. Several small chronic ischemic infarctions are noted in the left cerebral hemisphere; therefore, the latter is probably more likely.